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Research has characterised Tesamorelin’s visceral adipose biology in rodent and non-human primate lipodystrophy models and clinical research paradigms examining GH-driven visceral adipocyte lipolysis mechanisms, the selective visceral versus subcutaneous fat depot sensitivity to GH-mediated lipolysis, hepatic and peripheral IGF-1 axis contributions to the net visceral fat reduction outcome, and the temporal dynamics of visceral adipose tissue reduction under sustained Tesamorelin-driven GH secretory stimulation

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